SARS-CoV-2 spike protein interacts with and activates TLR41

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A Possible Role for Anti-idiotype Antibodies in SARS-CoV-2 Infection and Vaccination

Aptamer blocking S-TLR4 interaction selectively inhibits SARS-CoV-2 induced inflammation

IJMS, Free Full-Text

IJMS, Free Full-Text

IJMS, Free Full-Text

SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells

Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice

Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice

Differential Expression Patterns of Toll-like Receptors in COVID-19 Patients

SARS-CoV-2 spike protein induces inflammation via TLR2-dependent activation of the NF-κB pathway

SARS-CoV-2 nucleocapsid protein, rather than spike protein, triggers a cytokine storm originating from lung epithelial cells in patients with COVID-19

Cureus, A Potential Role of the Spike Protein in Neurodegenerative Diseases: A Narrative Review

SARS-CoV-2 Omicron Spike shows strong binding affinity and favourable interaction landscape with the TLR4/MD2 compared to other variants - ScienceDirect