SARS-CoV-2 spike protein interacts with and activates TLR41
A Possible Role for Anti-idiotype Antibodies in SARS-CoV-2 Infection and Vaccination
Aptamer blocking S-TLR4 interaction selectively inhibits SARS-CoV-2 induced inflammation
IJMS, Free Full-Text
IJMS, Free Full-Text
IJMS, Free Full-Text
SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells
Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice
Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice
Differential Expression Patterns of Toll-like Receptors in COVID-19 Patients
SARS-CoV-2 spike protein induces inflammation via TLR2-dependent activation of the NF-κB pathway
SARS-CoV-2 nucleocapsid protein, rather than spike protein, triggers a cytokine storm originating from lung epithelial cells in patients with COVID-19
Cureus, A Potential Role of the Spike Protein in Neurodegenerative Diseases: A Narrative Review
SARS-CoV-2 Omicron Spike shows strong binding affinity and favourable interaction landscape with the TLR4/MD2 compared to other variants - ScienceDirect