Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

Monomeric CRP contributes to complement control in fluid phase and on cellular surfaces and increases phagocytosis by recruiting factor H

Glomerular Disease of Complement Dysregulation (C3 Glomerulopathy, Atypical Hemolytic-Uremic Syndrome)

The role of the alternative pathway of complement activation in glomerular diseases

C3 glomerulopathies (C3G) are caused by dysregulation of the

The role of the alternative pathway of complement activation in glomerular diseases

Complement-mediated kidney diseases - ScienceDirect

Recurrent C3 glomerulopathy after kidney transplantation - ScienceDirect

Complement-mediated kidney diseases - ScienceDirect

Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

C3 glomerulopathy and atypical hemolytic uremic syndrome: an updated review of the literature on alternative complement pathway disorders

MAGI-2 orchestrates the localization of backbone proteins in the slit diaphragm of podocytes - Kidney International

Anti-complement-factor H-associated glomerulopathies

Regulation of regulators: Role of the complement factor H-related proteins - ScienceDirect

Proposed model for complement activation in hemodialysis (HD). The